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Download Molecular Mechanisms of Hypercoagulable States (Medical Intelligence Unit Series) eBook

by Andrew I. Schafer

Download Molecular Mechanisms of Hypercoagulable States (Medical Intelligence Unit Series) eBook
ISBN:
0412130718
Author:
Andrew I. Schafer
Category:
Medicine & Health Sciences
Language:
English
Publisher:
Chapman & Hall; 1 edition (January 1, 1997)
Pages:
212 pages
EPUB book:
1871 kb
FB2 book:
1320 kb
DJVU:
1723 kb
Other formats
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Rating:
4.8
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269


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Start by marking Molecular Mechanisms of Hypercoagulable States as Want to Read: Want to Read savin. ant to Read. It is now recognized that there are a number of specific genetic abnormalities, including specific mutations in antithrombotic genes, which cause inherited hypercoagulable states, also termed thrombophilias. This book assembles in one volume a complete and cohesive picture of our current understanding of the molecular basis of hypercoagulable states.

Molecular Mechanisms of Hypercoagulable States. by Andrew I. Schafer.

Patients are considered to have hypercoagulable states if they have laboratory abnormalities or clinical conditions that are . The number of specific primary hypercoagulable states that are recognized is growing.

Patients are considered to have hypercoagulable states if they have laboratory abnormalities or clinical conditions that are associated with an increased risk of thrombosis (prethrombotic states) or if they have recurrent thrombosis without recognizable predisposing factors (thrombosis-prone).

Molecular Mechanisms of Fanconi Anemia (Medical Intelligence Unit). Shamin I. Ahmad & Sandra H. Kirk (Ed. Download (pdf, . 0 Mb) Donate Read.

Molecular Mechanisms of Disseminated Intravascular Coagulation Medical Intelligence Unit Unnumbered.

This book attempts to dissect the complexity of the molecular mechanisms of insulin action with a special emphasis on those features of the system that are subject to alteration in type 2 diabetes and other insulin resistant states. It explores insulin action at the most basic levels, through complex systems.

Medical intelugence unit. Molecular Mechanisms of Phagocytosis Carlos Rosales, P. Universidad Nacional Autonoma de Mexico Mexico City, Mexico. L A n D e s b I o s C I e n C e, eurekah. springer science+business media new york, new york. p. ; cm. ~ (Medical intelligence unit) Includes bibliographical references and index. ISBN 0-387-25419-6 1. Phagocytosis. It explores insulin action at the most basic levels. Mechanisms of Insulin Action (Medical Intelligence Unit) Hardcover – June 27, 2007.

A hypercoagulable state, . Mechanisms of thrombosis in obesity. Basic Mechanisms and Pathogenesis of Venous Thrombosis. Blood Rev. 2009; 23(5): pp. 225–229. thrombophilia, is characterized by an increased predisposition to form blood clots. 2015; 20(5): pp. 437–444. 11. Tchaikovski SN, Rosing J. Mechanisms of estrogen-induced venous thromboembolism. Thromb Res. 2010; 126(1): pp. 5–11.

The molecular basis of specific inherited hypercoagulable states has been recently elucidated. With the description of resistance to activated protein C, which is the commonest coagulation defect associated with thrombophilia, a specific primary hypercoagulable state can be identified in over 50% of patients with thrombophilia. cle{ulableSM, title {Hypercoagulable states: molecular genetics to clinical practice}, author {AndrewI Schafer}, journal {The Lancet}, year {1994}, volume {344}, pages {1739-1742} }. AndrewI Schafer.

Presents the current scientific status of this rapidly moving area of biomedical research. Following an overview of the field, seven chapters provide detailed discussion of each of the primary hypercoagulable states, including antithrombin deficiency, protein C and protein S deficiency, disorders of fibrinogen, and the most recently described phenomenon of resistance to activated protein C. Additional chapters address the hypercoagulable state of cancer, the clinical evaluation of hypercoagulable states and the association of hemostatic factors with cardiovascular risk. Distributed in the US by Chapman & Hall. Annotation c. by Book News, Inc., Portland, Or.